| 1 |
Seymour CW, Liu VX, Iwashyna TJ, et al. Assessment of clinical criteria for sepsis: for the third international consensus definitions for sepsis and septic shock (Sepsis-3) [J]. JAMA, 2016, 315(8): 762-774.
|
| 2 |
Fleischmann C, Scherag A, Adhikari NK, et al. Assessment of global incidence and mortality of hospital-treated sepsis. current estimates and limitations [J]. Am J Respir Crit Care Med, 2016, 193(3): 259-272.
|
| 3 |
Pinheiro KHE, Azêdo FA, Areco KCN, et al. Risk factors and mortality in patients with sepsis, septic and non septic acute kidney injury in ICU [J]. Orgão oficial de Sociedades Brasileira e Latino-Americana de Nefrologia. 2019.
|
| 4 |
Pabla N, Gibson AA, Buege M, et al. Mitigat ion of acute kidney injury by cell-cycle inhibitors that suppress both CDK4/6 and OCT2 functions [J]. Proc Natl Acad Sci, 2015, 112(16): 5231-5236.
|
| 5 |
Morgan DO. Cyclin-dependent kinases: engines, clocks, and microprocessors [J]. Annu Rev Cell Dev Biol, 1997, 13: 261-291.
|
| 6 |
Criqui MC, Weingartner M, Capron A, et al. Sub-cellular localisation of GFP-tagged tobacco mitotic cyclins during the cell cycle and after spindle checkpoint activation [J]. Plant J, 2001, 28(5): 569-581.
|
| 7 |
Awasthi P, Foiani M, Kumar A. ATM and ATR signaling at a glance [J]. J Cell Sci, 2016, 129(6): 1285.
|
| 8 |
Dulić V, Kaufmann WK, Wilson SJ, et al. p53-dependent inhibition of cyclin-dependent kinase activities in human fibroblasts during radiation-induced G1 arrest [J]. Cell, 1994, 76(6): 1013-1023.
|
| 9 |
Russo AA, Tong L, Lee JO, et al. Structural basis for inhibition of the cyclin-dependent kinase Cdk6 by the tumour suppressor p16INK4a [J]. Nature, 1998, 395(6699): 237-243.
|
| 10 |
Yang QH, Liu DW, Long Y, et al. Acute renal failure during sepsis: Potential role of cell cycle regulation [J]. J Infect, 2009, 58(6): 459-464.
|
| 11 |
Iwakura T, Fujigaki Y, Fujikura T, et al. Acquired resistance to rechallenge injury after acute kidney injury in rats is associated with cell cycle arrest in proximal tubule cells [J]. Am J Physiol Renal Physiol, 2016, 310(9): F872-884.
|
| 12 |
Dirocco DP, Bisi J, Roberts P, et al. CDK4/6 inhibition induces epithelial cell cycle arrest and ameliorates acute kidney injury [J]. Am J Physiol Renal Physiol, 2014, 306(4): F379.
|
| 13 |
Pabla N, Gibson AA, Buege M, et al. Mitigation of acute kidney injury by cell-cycle inhibitors that suppress both CDK4/6 and OCT2 functions [J]. Proc Natl Acad Sci, 2015, 112(16): 5231-5236.
|
| 14 |
Cuartero M, Ballús J, Sabater J, et al. Cell-cycle arrest biomarkers in urine to predict acute kidney injury in septic and non-septic critically ill patients [J]. Ann Intensive Care, 2017, 7(1): 92.
|
| 15 |
Yang QH, Liu DW, Long Y, et al. Acute renal failure during sepsis: potential role of cell cycle regulation [J]. J Infect, 2009, 58(6): 459-464.
|
| 16 |
Aydoğdu M, Boyacı N, Yüksel S, et al. A promising marker in early diagnosis of septic acute kidney injury of critically ill patients: urine insulin like growth factor binding protein-7 [J]. Scand J Clin Lab Invest, 2016, 76(5): 402-10.
|
| 17 |
Gu X, Peng CY, Lin SY, et al. P16INK4a played a critical role in exacerbating acute tubular necrosis in acute kidney injury [J]. Am J Transl Res, 2019, 11(6): 3850-3861.
|
| 18 |
Higgins SP, Tang Y, Higgins CE, et al. TGF-β1/p53 signaling in renal fibrogenesis [J]. Cell Signal, 2018, 43: 1-10.
|
| 19 |
Ying Y, Kim J, Westphal SN, et al. Targeted deletion of p53 in the proximal tubule prevents ischemic renal injury [J]. J Am Soc Nephrol, 2014, 25(12): 2707-16.
|
| 20 |
Peng ZY, Zhou F, Kellum JA. Cross-species validation of cell cycle arrest markers for acute kidney injury in the rat during sepsis [J]. Intensive Care Med Exp, 2016, 4(1): 12.
|
| 21 |
McCullough PA, Ostermann M, Forni LG, et al. Serial urinary tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 and the prognosis for acute kidney injury over the course of critical illness [J]. Cardiorenal Med, 2019, 9(6): 358-369.
|
| 22 |
张卉, 杨晓. 尿[TIMP-2]×[IGFBP-7]对心脏术后患者急性肾损伤早期预测价值的Meta分析 [J]. 临床肾脏病杂志, 2019, 19(5) : 340-346.
|
| 23 |
Wang X, Ma T, Wan X, et al. IGFBP7 regulates sepsis-induced acute kidney injury through ERK1/2 signaling [J]. Acta Biochim Biophys Sin, 2019, 51(8): 799-806.
|
| 24 |
Wang Z, Famulski K, Lee J, et al. TIMP2 and TIMP3 have divergent roles in early renal tubulointerstitial injury [J]. Kidney Int, 2014, 85(1): 82-93.
|
| 25 |
Vijayan A, Faubel S, Askenazi DJ, et al. Clinical use of the urine biomarker [TIMP-2]×[IGFBP7] for acute kidney injury risk assessment [J]. Am J Kidney Dis, 2016, 68(1): 19-28.
|
| 26 |
El Minshawy O, Khedr M, Youssuf A, et al. Value of the cell cycle arrest biomarkers in the diagnosis of pregnancy related acute kidney injury [J]. Biosci Rep, 2020, BSR20200962.
|
| 27 |
Hatton GE, Wang YW, Isbell KD, et al. Urinary cell cycle arrest proteins urinary tissue inhibitor of metalloprotease 2 and insulin-like growth factor binding protein 7 predict acute kidney injury after severe trauma: A prospective observational study [J]. J Trauma Acute Care Surg, 2020, 89(4): 761-767.
|
| 28 |
Wang K, Xie S, Xiao K, et al. Biomarkers of sepsis-induced acute kidney injury [J]. Biomed Res Int, 2018: 6937947.
|
| 29 |
Jia HM, Huang LF, Zheng Y, et al . Prognostic value of cell cycle arrest biomarkers in patients at high risk for acute kidney injury: A systematic review and meta-analysis [J]. Nephrology (Carlton), 2017, 22(11): 831-837.
|
| 30 |
Nusshag C, Rupp C, Schmitt F, et al. Cell cycle biomarkers and soluble urokinase-type plasminogen activator receptor for the prediction of sepsis-induced acute kidney injury requiring renal replacement therapy: a prospective, exploratory study [J]. Crit Care Med, 2019, 47(12): e999-e1007.
|
| 31 |
Lin Z, Liu Z, Wang X, et al. MiR-21-3p plays a crucial role in metabolism alteration of renal tubular epithelial cells during sepsis associated acute kidney injury via AKT/CDK2-FOXO1 pathway [J]. Biomed Res Int, 2019:2821731.
|
| 32 |
Li F, Liu Z, Tang C, et al. FGF21 is induced in cisplatin nephrotoxicity to protect against kidney tubular cell injury [J]. FASEB J, 2018, 32(6): 3423-3433.
|