1 |
Beesley SJ, Weber G, Sarge T, et al. Septic Cardiomyopathy[J]. Crit Care Med, 2018, 46(4): 625-634.
|
2 |
Kakihana Y, Ito T, Nakahara M, et al. Sepsis-induced myocardial dysfunction: pathophysiology and management[J]. J Intensive Care, 2016, 4: 22.
|
3 |
Fu S, Ping P, Zhu Q, et al. Brain Natriuretic Peptide and Its Biochemical, Analytical, and Clinical Issues in Heart Failure: A Narrative Review[J]. Front Physiol, 2018, 9: 692.
|
4 |
Papanikolaou J, Makris D, Mpaka M, et al. New insights into the mechanisms involved in B-type natriuretic peptide elevation and its prognostic value in septic patients[J]. Crit Care, 2014, 18(3): R94.
|
5 |
Januzzi JL, Tournoux AAC, Christenson RH, et al. N-Terminal Pro-B-Type Natriuretic Peptide in the Emergency Department: The ICON-RELOADED Study[J]. J Am Coll Cardiol, 2018, 71(11): 1191-1200.
|
6 |
Pulido JN, Afessa B, Masaki M, et al. Clinical spectrum, frequency, and significance of myocardial dysfunction in severe sepsis and septic shock[J]. Mayo Clin Proc, 2012, 87(7): 620-628.
|
7 |
Varpula M, Pulkki K, Karlsson S, et al. Predictive value of N-terminal pro-brain natriuretic peptide in severe sepsis and septic shock[J]. Crit Care Med, 2007, 35(5): 1277-1283.
|
8 |
Witthaut R, Busch C, Fraunberger P, et al. Plasma atrial natriuretic peptide and brain natriuretic peptide are increased in septic shock: impact of interleukin-6 and sepsis-associated left ventricular dysfunction[J]. Intensive Care Med, 2003, 29(10): 1696-1702.
|
9 |
Tung, R.H., Garcia C., Morss A.M., et al. Utility of B-type natriuretic peptide for the evaluation of intensive care unit shock[J]. Crit Care Med, 2004, 32(8): 1643-1647.
|
10 |
Pirracchio R, Deye N, Lukaszewicz AC, et al. Impaired plasma B-type natriuretic peptide clearance in human septic shock[J]. Crit Care Med, 2008, 36(9): 2542-2546.
|
11 |
Rubini Gimenez M, Twerenbold R, Reichlin T, et al. Direct comparison of high-sensitivity-cardiac troponin I vs. T for the early diagnosis of acute myocardial infarction[J]. Eur Heart J, 2014, 35(34): 2303-2311.
|
12 |
van der Linden N, Wildi K, Twerenbold R, et al. Combining high-sensitivity cardiac troponin I and cardiac troponin T in the early diagnosis of acute myocardial infarction[J]. Circulation, 2018, 138(10): 989-999.
|
13 |
Roffi M, Patrono C, Collet JP, et al. 2015 ESC Guidelines for the management of acute coronary syndromes in patients presenting without persistent ST-segment elevation: task force for the management of acute coronary syndromes in patients presenting without persistent ST-segment elevation of the European Society of Cardiology (ESC)[J]. Eur Heart J, 2016, 37(3): 267-315.
|
14 |
du Fay de Lavallaz J, Badertscher P, Nestelberger T, et al. B-Type natriuretic peptides and cardiac troponins for diagnosis and risk-stratification of syncope[J]. Circulation, 2019 Feb 25.
|
15 |
Goldstein SA, Newby LK, Cyr DD, et al. Relationship between peak troponin values and long-term ischemic events among medically managed patients with acute coronary syndromes[J]. J Am Heart Assoc, 2017, 6(4): e005334.
|
16 |
Blankenberg S, Salomaa V, Makarova N, et al. Troponin I and cardiovascular risk prediction in the general population: the BiomarCaRE consortium[J]. Eur Heart J, 2016, 37(30): 2428-2437.
|
17 |
Liu Z, Cui L, Wang Y, et al. Cardiac troponin I and ventricular arrhythmia in patients with chronic heart failure[J]. Eur J Clin Invest, 2006, 36(7): 466-472.
|
18 |
Welsh P, Preiss D, Hayward C, et al. Cardiac troponin T and troponin I in the general population[J]. Circulation, 2019, 139(24): 2754-2764.
|
19 |
Wright RS, Williams BA, Cramner H, et al. Elevations of cardiac troponin I are associated with increased short-term mortality in noncardiac critically ill emergency department patients[J]. Am J Cardiol, 2002, 90(6): 634-636.
|
20 |
Cheng H, Fan WZ, Wang SC, et al. N-terminal pro-brain natriuretic peptide and cardiac troponin I for the prognostic utility in elderly patients with severe sepsis or septic shock in intensive care unit: A retrospective study[J]. J Crit Care, 2015, 30(3): 654 e659-614.
|
21 |
Maeder M, Fehr T, Rickli H, et al. Sepsis-associated myocardial dysfunction: diagnostic and prognostic impact of cardiac troponins and natriuretic peptides[J]. Chest, 2006, 129(5): 1349-1366.
|
22 |
Landesberg G, Jaffe AS, Gilon D, et al. Troponin elevation in severe sepsis and septic shock: the role of left ventricular diastolic dysfunction and right ventricular dilatation[J]. Crit Care Med, 2014, 42(4): 790-800.
|
23 |
Jeremias A, Gibson CM. Narrative review: alternative causes for elevated cardiac troponin levels when acute coronary syndromes are excluded[J]. Ann Intern Med, 2005, 142(9): 786-791.
|
24 |
Ammann, P., Fehr T., Minder E.I., et al. Elevation of troponin I in sepsis and septic shock[J]. Intensive Care Med, 2001, 27(6): 965-969.
|
25 |
Favory R, Neviere R. Significance and interpretation of elevated troponin in septic patients[J]. Crit Care, 2006, 10(4): 224.
|
26 |
Masson S, Caironi P, Fanizza C, et al. Sequential N-Terminal Pro-B-Type natriuretic peptide and high-sensitivity cardiac troponin measurements during albumin replacement in patients with severe sepsis or septic shock[J]. Crit Care Med, 2016, 44(4): 707-716.
|
27 |
Otaki Y, Watanabe T, Kubota I. Heart-type fatty acid-binding protein in cardiovascular disease: A systemic review[J]. Clin Chim Acta, 2017, 474: 44-53.
|
28 |
Liebetrau C, Nef HM, Dörr O, et al. Release kinetics of early ischaemic biomarkers in a clinical model of acute myocardial infarction[J]. Heart, 2014, 100(8): 652-657.
|
29 |
Carroll C, Al Khalaf M, Stevens JW, et al. Heart-type fatty acid binding protein as an early marker for myocardial infarction: systematic review and meta-analysis[J]. Emergency Medicine Journal, 2013, 30(4): 280-286.
|
30 |
Kellens S, Verbrugge FH, Vanmechelen M, et al. Point-of-care heart-type fatty acid binding protein versus high-sensitivity troponin T testing in emergency patients at high risk for acute coronary syndrome[J]. Eur Heart J Acute Cardiovasc Care, 2016, 5(2): 177-184.
|
31 |
Young JM, Pickering JW, George PM, et al. Heart Fatty Acid Binding Protein and cardiac troponin: development of an optimal rule-out strategy for acute myocardial infarction[J]. BMC Emerg Med, 2016, 16(1): 34.
|
32 |
Fan TT, Feng XY, Yang YZ, et al. Downregulation of PI3K-gamma in a mouse model of sepsis-induced myocardial dysfunction[J]. Cytokine, 2017, 96: 208-216.
|
33 |
Zhou G, Ye L, Zhang L, et al. Association of myeloid cells of triggering receptor-1 with left ventricular systolic dysfunction in BALB/c mice with sepsis[J]. Mediators Inflamm, 2014, 2014: 391492.
|
34 |
Tao F, Peng L, Li J, et al. Association of serum myeloid cells of soluble triggering receptor-1 level with myocardial dysfunction in patients with severe sepsis[J]. Mediators Inflamm, 2013, 2013: 819246.
|
35 |
Hagiwara S, Iwasaka H, Uchino T, et al. High mobility group box 1 induces a negative inotropic effect on the left ventricle in an isolated rat heart model of septic shock: a pilot study[J]. Circ J, 2008, 72(6): 1012-1017.
|
36 |
McLean AS, Huang SJ, Salter M. Bench-to-bedside review: the value of cardiac biomarkers in the intensive care patient[J]. Crit Care, 2008, 12(3): 215.
|
37 |
Wang H, Bei Y, Shen S, et al. miR-21-3p controls sepsis-associated cardiac dysfunction via regulating SORBS2[J]. J Mol Cell Cardiol, 2016, 94: 43-53.
|
38 |
Wang B, Chen G, Li J, et al. Neutrophil gelatinase-associated lipocalin predicts myocardial dysfunction and mortality in severe sepsis and septic shock[J]. Int J Cardiol, 2017, 227: 589-594.
|
39 |
Zhang Z, Dai H, Yu Y, et al. Elevated pregnancy-associated plasma protein A predicts myocardial dysfunction and death in severe sepsis[J]. Ann Clin Biochem, 2014, 51(Pt 1): 22-29.
|
40 |
Foussas SG, Zairis MN, Makrygiannis SS, et al. The significance of circulating levels of both cardiac troponin I and high-sensitivity C reactive protein for the prediction of intravenous thrombolysis outcome in patients with ST-segment elevation myocardial infarction[J]. Heart, 2007, 93(8): 952-956.
|
41 |
Peacock F, Morris DL, Anwaruddin S, et al. Meta-analysis of ischemia-modified albumin to rule out acute coronary syndromes in the emergency department[J]. Am Heart J, 2006, 152(2): 253-262.
|